Personal Information:
Name: Min Kong
Gender: Female
Date of birth: 1978-04
Place of birth: Qufu, Shandong, China
Nationality: China
Marital status: Married
Department: Department of Neurology, Yantaishan Hospital, Shandong Province, 264000, China
Address: No.91, Jie fang Road , Yantai City , Shandong Province, China
E-mail: kk_kmm@sina.com
Education:
2006~2009, Ph.D. Shanghai jiaotong University, Shanghai, China
2002~2005, M.D. Shanghai University of Traditional Chinese Medicine, Shanghai, China
Employment:
2016~present Associate chief physician, Dept. of Neurology, Yantaishan Hospital, Yantai City, Shandong, P.R.China
2013~present, Attending doctor, Dept. of Neurology, Yantaishan Hospital, Yantai City, Shandong, P.R.China
2009~2012, Resident doctor, Dept. of Neurology, Yantaishan Hospital, Yantai City, Shandong, P.R.China
Clinical Service Area
Neurology; Acupuncture and moxibustion
Specialty
Movement Disorders
Parkinson's Disease
Tremors;
Focal dystonia;
Program control of deep brain stimulation
Hemiplegia after stroke
All kinds of pain
Obesity
Honors and Awards:
The second prize of Yantai scientific and Technological Progress Award (2015)
The second prize of Yantai scientific and Technological Progress Award (2013)
Medical science and technology award of Shanghai City (2009)
Current Research Interests:
Clinical study of effectiveness and safety of rasagiline mesylate orally disintegrating tablets in the treatment of Parkinson’s disease patients with motor fluctuations.
Mitochondrial mechanism in the Parkinson’s disease and Alzheimer's disease and the related neuroprotective effects.
Synaptic plasticity and Parkinson’s disease and levodopa-induced motor complications. Focus on NMDA and AMPA signaling complex and synaptic plasticity.
Clinical study of effectiveness of deep brain stimulation in the treatment of moderate and advanced Parkinson’s disease.
Clinical study of effectiveness of Botox in the treatment of focal dystonia, such as spasmodic torticollis, facial spasm.
Clinical study of effectiveness of Acupuncture and moxibustion in the treatment of hemiplegia after stroke, all kinds of pain, depression and anxiety, obesity, insomnia, and so on.
Publications:
[1] Ba M, Kong M, Ma G. Postsynaptic density protein 95 regulated
NR2B tyrosine
phosphorylation and interactions of Fyn with NR2B
in levodopa-induced dyskinesia rat model. Drug Des Devel Ther.2015, 9:199-206.
[2] Kong M, Ba M. et.al. NR2B antagonist CP-101,606 inhibits NR2B phosphorylation at tyrosine-1472 and its interactions with Fyn in levodopa-induced dyskinesia rat model. Behavioural Brain Research. 2015, 282:46–53.
[3]
Kong M, Ba M. Protective effects of diazoxide against Aβ
25-35-induced PC12 cell apoptosis due to prevention of endoplasmic reticulum stress. Neuroreport. 2012; 23(8):493-7.
[4] Kong M, Ba M, Liang H, Ma L, Yu Q, Yu T, Wang Y. 5’-aza-dC sensitizes paraquat toxic effects on PC12 cell. Neurosci Lett. 2012, 524(1):35-39.
[5] Ba M,
Kong M, Yu G, et al. GluR1 phosphorylation and persistent expression of levodopa-induced motor response alterations in the Hemi-Parkinsonian rat. Neurochem Res. 2011, 36(6):1135-1144.
[6] Kong M, Ba MW (co-first author), Song L,
et al. Comparative effects of acute or chronic administration of levodopa to 6-OHDA-lesioned rats on the expression and phosphorylation of N-methyl-D-aspartate Receptor NR1 subunits in the striatum. Neurochem Res. 2009,34:1513–1521.
[7] Maowen Ba,
Min Kong, Guozhao Ma,
et al. Cellular and behavioural effects of 5-HT1A receptor agonist 8-OH-DPAT in a rat model of levodopa-induced motor complications. Brain Research. 2007,1127(1):177-184.
[8] Maowen Ba
, Min Kong, Hongqi Yang,
et al. Changes in Subcellular Distribution and Phosphorylation of GluR1 in Lesioned Striatum of 6-OHDA-Lesioned and L-dopa-Treated Rats. Neurochem Res. 2006, 31(11):1337-1347.
[9] Maowen Ba,
Min Kong. Clinical progress of Parkinson disease and motor complications. Chemical Industry Press. 2012. Peking. ISBN:978-7-122-13553-7.
[10] Lu Song,
Min Kong, Yaping Ma, et al.Inhibitory Effect of 8-(3-chlorostryryl) Caffeine on Levodopa-induced Motor Complications Is Associated with Intracellular Signaling Pathway in 6-OHDA-Lesioned Rats.Brain Research. 2009.
Grants:
The Young Scholars General Program of National Natural Science Foundation of Shandong province, China (Project proposal No. ZR2013HQ003). (The first responsible person, 2013-2016).
Medical science and technology development plans of Shandong province, China (Project proposal No. 2014WS0260). (The first responsible person, 2014-2016).
The science and technology development plan item of Yantai city, Shandong province, China (Project proposal No. 2014WS035). (The first responsible person, 2014-2016).